The forest of spikes, revisited - pulmonary embolism in the elderly. FDA report, no action planned on their part re the jabs.
What to do? EDTA chelation is being used successfully by Ana Maria Mihalcea, MD, PhD.
From Exposing the Darkness: BREAKING: FDA Now Says Pfizer’s COVID-19 VACCINE Linked to Blood Clotting… (substack.com)
“The study found that the vaccines are also linked to a lack of oxygen to the heart, a blood platelet disorder called immune thrombocytopenia, and another type of clotting called intravascular coagulation.
More in-depth evaluations, such as comparisons with populations who received influenza vaccines, showed those three as no longer meeting the statistical threshold for a signal. Researchers looked at data covering 17.4 million elderly Americans who received a total of 34.6 million vaccine doses between December 10, 2020, and January 16, 2022.
The study was published in the journal Vaccine.”
Remember the statistical muffling of adverse events by comparing them to each other, expecting one odd thing to stand out. With the chimeric spike we have a forest of things standing out from any normal treatment or human clinical trial. By comparing the forest to the forest, it all looks about level - no problems here. See the Steve Kirsch post/discussion in this: The Culling? It is for the greater good? (substack.com)
Aka “more in-depth evaluations,” the placebo game - comparisons with influenza jab recipients - might show the same adverse forest of spikes if it is being monkeyed with too, as has been suggested but I couldn’t link to anything specific. That is the placebo game - compare an experimental product to something that is not inert like a starch or sugar pill or saline solution, instead use something known to cause similar adverse effects so the the experimental product won’t look bad in comparison.
So, what we likely have here is a very tall tree in the pulmonary embolism column, and three not quite as tall, tall trees in the 1) “a lack of oxygen to the heart,” 2) “a blood platelet disorder called immune thrombocytopenia, and” 3) “another type of clotting called intravascular coagulation.” From BREAKING: FDA Now Says Pfizer’s COVID-19 VACCINE Linked to Blood Clotting… (substack.com).
Those four trees are just a little taller than the rest of the forest of adverse effects that are being called climate change, or duvet shaking (caution with those volatile chemicals). The FDA is not acting on this information and haven’t acted on Steve Kirsch’s prompting or challenges. We the people need to protect ourselves and each other because our government seems complicit.
My placebo series on peace-is-happy.org has a couple posts that mention the experimental abuse of ‘inert’ placebos. Experimental treatment adverse effects are compared to those of an 'Inert' Placebo in research. (peace-is-happy.org) The post includes an example from a book - statin research had almost identical rates of adverse effects between the experimental and placebo in numerous studies, with placebo adverse effects ranging from 2.7% to 80.4%. From: Dr. Malcolm Kendrick, The Great Placebo Scandal. The “placebo effect” average, over a long history of experimental design is about 40% - generally we can expect 40% of people to ‘feel better’ taking the sugar pill that the authority figure in a white coat handed them because they said it would make them feel better.
The placebo effect is psychological and is related to dopamine levels. Some people have more and are more susceptible to the effect. See: Placebo and Nocebo Effects and COMT gene alleles. (peace-is-happy.org)
The placebo effect can also be negative - the nocebo voodoo curse that can kill a believer. In the case of an experiment if the participants are told at the beginning to report any changes in their health that might be a side effect, and are led with words like, “such as upset stomach, diarrhea, or a headache.” We can expect about 40% of the participants to report one of those three things. What we would not expect is for 2.7% or 80.4% of the participants to report one of those three things.
The placebo effect is not supposed to be caused by an active agent that can physically do something. A voodoo curse can’t really kill you. An experiment that has an ‘inert’ placebo that causes an 80.4% adverse reaction rate is just really odd. The kiss on the forehead by mom that makes you feel better as a child is more of a placebo but may be doing something via emotional connection and increased oxytocin in the child.
On the ‘what to do’ front - EDTA chelation therapy is working for Ana Maria Mihalcea, MD, PhD with her patients. This post has images of the nanotech like things that form within 2 days of a vial being examined.
Otherwise, I take serrapeptase every night, drink pomegranate/dandelion tea and take other supplements to prevent inflammation and support mitochondria.
I also avoid glyphosate. Our symbionts, our bacterial mitochondria, need us to stop using glyphosate. That seems like simple science - glyphosate was developed as an antibiotic and mineral chelator before being shifted to agricultural use. An antibiotic and mineral chelator should not be sprayed on our food supply.
List of glyphosate posts is in this post and is more the topic of the post: The last post got a big edit and has resource links now. (Niacin/wild hamster cannibalism/glyphosate.) (substack.com)
We are being poisoned in many ways and the illness has been normalized for a long time. Humans do not suddenly become obese on average within two decades. It is the glyphosate and lack of iodine, and excess bromide, fluoride.
What to do? Grow some food. Network locally. Build teams that care about health in your real world.
Photo op
Disclaimer: This information is provided for educational purposes within the guidelines of Fair Use. It is not intended to provide individual guidance. Please seek a health care provider for individualized health care guidance.
Question oh wise one!
Is taking trans resvetrol 100 mg/ d safe as antioxidant or is low dose a prooxidant? Protumor? Proestrogen etcetc
I started it this week adding to other antioxidants intake but wonder if should stop after reading this?!
What do yr studies show?
"A study carried out by Martins et al. revealed that resveratrol can modulate different pathways at a time, which can result in distinct and even opposite biological effects, depending on its concentration or treatment time defined. The authors documented that, although a dose-dependent resveratrol pro-oxidative effect leads to cells oxidative stress over lesser time exposure, at same dose but with an increase in exposure time, less expressive cytotoxicity was found. This suggest that surviving cells seemed to be more resistant to resveratrol-induced damages, being its effects attenuated over treatment time [114]. Additionally, low resveratrol doses (0.1–1.0 μg/mL) has been documented to enhance cell proliferation, whereas higher doses (10.0–100.0 μg/mL) induces apoptosis (Figure 2) and decreases mitotic activity on human tumors and endothelial cells [122]. Recently, dual resveratrol pattern effects on HT-29 colon cancer cells death and proliferation were observed, where at low concentrations (1 and 10 μmol/L), resveratrol increased cells number, while at higher doses (50 or 100 μmol/L) resveratrol reduced cells number and increased apoptotic or necrotic cells percentage [123]
There is an interesting correlation among dietary polyphenols pro-oxidant and cytotoxic activities, such as to resveratrol. In fact, since every antioxidant is a redox agent it might become a pro-oxidant, accelerating lipid peroxidation and/or inducing DNA damages under special conditions. In this way, it has been proposed that such pro-oxidant action could be an important mechanism of action to resveratrol anti-cancer and apoptotic-inducing properties [112]. It has already been reported that resveratrol can lead to DNA damages, as well as to a reversible or irreversible cell cycle interruption mediated by its pro-oxidant effect [117]. Recently, Plauth et al. [125] proposed that cellular response to resveratrol treatment is based on oxidative triggering action, that can lead to cell fitness hormetic induction towards a more reductive state, so as to physiological resilience raising in fight oxidative stress. Also, it has been reported earlier that a critical balance between intracellular hydrogen peroxide (H2O2) and O2– decides cells fate to ...
Besides, in absence of estrogen (E2), resveratrol exerts mixed estrogen agonist/antagonist activities in some mammary cancer cell lines, but in the presence of E2, resveratrol acts as an anti-estrogen [134]. In another report, it was demonstrated that resveratrol abolishes serum deprivation-induced elevated caspase 3 activity, suggesting its rescue effect via p38 MAPK signaling [135]. Resveratrol also regulates mitochondrial respiratory chain function, with mitochondrial complex I (CI) as a direct target of this molecule. It was also in vivo demonstrated that, in young and old mice brain mitochondria, resveratrol increased CI, while in aged animals with low antioxidant defenses led to oxidative stress. Therefore, not only dose, but also age at the time of treatment, can modulate intracellular and mitochondrial redox status, switching from resveratrol beneficial to deleterious effects, highlighting the importance of a balance between resveratrol pro- and antioxidant effects, that depends on its dose and age as well [136]. Yang et al. [137] reported dual resveratrol roles in pancreatic cancer cells: one as a tumor suppressor through Bax up-regulation, and the other one as a tumor activator through VEGF-B up-regulation; so, resveratrol anticancer effect is much stronger than cancer promotion effect.
All the above highlighted studies show the pivotal role of dose-dependency and aging in resveratrol-induced responses towards health benefits. Also, in another study, aiming to compare resveratrol effects on aging-induced and re-nutrition-induced insulin resistance and its consequences on arterial system, the authors found that resveratrol improved insulin sensitivity in old mice fed standard diet, while did not improve insulin resistance status in old mice receiving high-protein diets. In contrast, resveratrol exhibited deleterious effects by increasing inflammation state and superoxide production and decreasing aortic distensibility. This data demonstrates that resveratrol seemed to be beneficial to malnourished state of physiological aging, whereas when associated with high protein diets in old mice, may increase atherogenesis-associated risk factors by triggering vascular alterations that could represent an additional risk factor for cardiovascular system