Unusual spike in use of children's acetominophen in Canada created shortages - in 2022.
The children's medication didn't run out during the "Covid Pandemic" but it is now. Why? asks Sheldon Yakiwchuk.
Why the sudden shortage in a child’s flu and pain killing drug - almost three years after the alleged pandemic - asks a Substack author, Sheldon Yakiwchuk.
What the actual F is going on here? Inflammatory changes due to an over-activation of vitamin A and carotenoids to Retinoic Acid possibly - would be my answer or part of it.
*Addition - I am in no way trying to be critical or rude or anything negative - pain really hurts, the kids are probably in pain. Science should not be a competition, it should be about finding solutions. Not just watching the details of how people are dying. Whether they got jabbed or not, the spike toxin is likely causing hyperinflammation and may have led to the over activation of Retinoic Acid problem, which would mean any meal with tomatoes, carrots, peaches, or sweet potato or liver or vitamin A fortified milk, would all be adding to the children’s flu-like body aches and other chronic low-level symptoms of inflammation. I found and bought a long review article on Myalgic Encephalomyelitis/Chronic Fatigue Syndrome. Retinoid metabolism is definitely involved but the review team says that aspect has not been studied much yet. *Pdf in my Dropbox is in the reference list if you want to read it. (5) Simple reminder - the point of learning more about what is wrong, is so you can figure out how to make things better again. Not to endlessly point out how wrong things are. Yes - our governments apparently are extremely stupid, or extremely evil. Okay, what next? There are cheaper and less painful ways to die if going along with being culled is an okay goal.
Exosomes from CoV jabbed parents or teachers can passively spread to children or others and if there are surface marker proteins that match, the genetic material may be transcribed by the passively exposed person’s cells and produce chimeric spike protein or possibly the S1 subunit. The graphene oxide content may also be passively shared and add to health consequences but not enough is known.
“Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) is common in post-acute sequelae of SARS-CoV-2 infection (PASC): Results from a post-COVID-19…” (4)
The short answer to What the F is going on?:
*I am in a hurry - this IS THE ANSWER though. Please listen. Our children need effective help, not ongoing denial of nutrition informaton.
The immune challenge of SARS-CoV2 and/or chimeric spike, seems to lead to an ongoing conversion of inactive vitamin A and carotenoids to the active Retinoic Acid, similar to what has been seen with Chronic Fatigue Syndrome following Epstein Barr Virus. Too much Retinoic Acid causes an inflammatory immune response that resembles fighting an intracellular virus - flu like body aches and congestion, brain fog and fatigue can be the daily result, affectionately known as ME/CFS (instead of as Vitamin A and Carotenoid Toxicity).
Retinoic Acid controls gene transcription for viral immune challenges and other purposes. It affects which genes are transcribed or not into proteins. In fibromyalgia patients (Nociceptive pain - My Everything Hurt! post) it was found that patients reporting worse pain did have significant differences in what genes were being transcribed or were downregulated - inactivated. The overall pattern appeared to be that the patients were fighting a viral infection - an intracellularly based one.
During my recent two-day Peach Flare up I was using 600 mg of ibuprofen several times a day because my puffy pain was so severe, and sudden onset. There had been no significant exercise to cause pain. I just ate a quarter of a peach, after having identified that I do seem to over-convert carotenoids to Retinoic Acid.
I was frankly astonished by the level of pain that developed in just a day and half after eating the peach. It took another night’s sleep after that to feel more normal again.
If these kids (or others) are also now suddenly over-converting vit A and carotenoids to Retinoic Acid, then they are in for daily pain because our diet has a wide range of sources. It is fairly difficult to avoid most rich sources of vitamin A and carotenoids - but the health difference is the difference between feeling fairly normal and feeling like you are fighting a flu or bad cold.
I added a couple replies:
Retinoid Toxicity as a result of a liver enzyme change is part of the problem in my opinion and experience.
Even if there was resistance to jabbing kids, the passive exposure from jabbed parents, and others, is a risk. It can cause a similar chimeric spike related low level lingering illness. My document Exosomes has an overview and some earlier reference links. I haven't added any in a while.
Retinoid Toxicity can be an ongoing change that causes more inflammation and pain. I don't usually need ibuprofen anymore but during two days of Retinoic Acid overdose (a quarter of a peach and a tiny bit of carrot) I did use the anti-inflammatory amount of 600 mg several times. From zero to nine capsules just because the farm market peach looked so good that I forgot to not eat any. Vak injury or viral infection including Sars2 potentially can cause the Chronic Fatigue Syndrome type of symptoms chronically afterwards, because the liver is over-activating all vitamin A or carotenoids to the active Retinoic Acid form - which can change which proteins are being transcribed and lead to a "fight virus" inflamed state. Flu like body aches. My third in a series of posts on the two day illness flair-up - just because I ate more vitamin A foods.
*I had Epstein Barr Virus as a teen and Chronic Fatigue like symptoms forever after that, ups and downs.
Disclaimer: Opinions are my own and the information is provided for educational purposes within the guidelines of fair use. While I am a Registered Dietitian this information is not intended to provide individual health guidance. Please see a health professional for individual health care purposes.
Tate W, Walker M, Sweetman E, Helliwell A, Peppercorn K, Edgar C, Blair A, Chatterjee A. Molecular Mechanisms of Neuroinflammation in ME/CFS and Long COVID to Sustain Disease and Promote Relapses. Front Neurol. 2022 May 25;13:877772. doi: 10.3389/fneur.2022.877772. PMID: 35693009; PMCID: PMC9174654. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9174654/
Wong TL, Weitzer DJ. Long COVID and Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS)-A Systemic Review and Comparison of Clinical Presentation and Symptomatology. Medicina (Kaunas). 2021 Apr 26;57(5):418. doi: 10.3390/medicina57050418. PMID: 33925784; PMCID: PMC8145228.
Friedman KJ, Murovska M, Pheby DFH, Zalewski P. Our Evolving Understanding of ME/CFS. Medicina (Kaunas). 2021 Feb 26;57(3):200. doi: 10.3390/medicina57030200. PMID: 33652622; PMCID: PMC7996879. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7996879/
Neurological And Psychiatric Risk Trajectories After SARS-CoV-2 Infection: An Analysis Of 2-Year Retrospective Cohort Studies Including 1 284 437 Patients. American Myalgic Encephalomyelitis and Chronic Fatigue Syndrome Society https://ammes.org/2022/08/18/neurological-and-psychiatric-risk-trajectories-after-sars-cov-2-infection-an-analysis-of-2-year-retrospective-cohort-studies-including-1%e2%80%88284%e2%80%88437-patients/
G. Anderson, M. Maes, Mitochondria and immunity in chronic fatigue syndrome, Progress in Neuro-Psychopharmacology and Biological Psychiatry, Volume 103, 2020, 109976, ISSN 0278-5846, https://doi.org/10.1016/j.pnpbp.2020.109976.
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