Nice touch, and they were doing so well with the other herbals🤦♂️
"The c-Myc oncogene is overexpressed in the majority of human cancers and contributes to the cause of at least 40% of tumors37. It encodes a helix-loop-helix leucine zipper transcription factor that dimerizes with its partner protein, Max, to transactivate gene expression."
And yes as I suspected, pharma have got us covered here via the borrowed, cleavable inserts:
Role of 14-3-3epsilon, c-Myc/Max, and Akt phosphorylation in HIV-1 gp 120-induced mesangial cell proliferation
...In parallel to the modulation of cell growth, gp 120 at low concentrations resulted in an increase in the expression of c-Myc, Max, and 14-3-3epsilon proteins and phosphorylation of ATP-dependent tyrosine kinases (Akt) at Ser(473). However, the expression of these proteins decreased with increasing concentrations of gp 120.
Nice touch, and they were doing so well with the other herbals🤦♂️
"The c-Myc oncogene is overexpressed in the majority of human cancers and contributes to the cause of at least 40% of tumors37. It encodes a helix-loop-helix leucine zipper transcription factor that dimerizes with its partner protein, Max, to transactivate gene expression."
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3505847/
And yes as I suspected, pharma have got us covered here via the borrowed, cleavable inserts:
Role of 14-3-3epsilon, c-Myc/Max, and Akt phosphorylation in HIV-1 gp 120-induced mesangial cell proliferation
...In parallel to the modulation of cell growth, gp 120 at low concentrations resulted in an increase in the expression of c-Myc, Max, and 14-3-3epsilon proteins and phosphorylation of ATP-dependent tyrosine kinases (Akt) at Ser(473). However, the expression of these proteins decreased with increasing concentrations of gp 120.
https://pubmed.ncbi.nlm.nih.gov/11208609/
Good to KNOW Jen!
THANK YOU for doing research.
Very welcome and thanks for the feedback!