Mpox part 4 - Geert says no one should be getting the Mpox jabs, roughly.

And more natural health aids via Pat on Telegram, thanks!

Phytonutrients that might help against Monkeypox include Rosmarinic acid, which might be most effective based on computer modeling. Others that may also be potent as inhibitors of Monkeypox virus include myricitrin, quercitrin and ofloxacin were also potentially good inhibitors, (Hu, et al., 2023), and resveratrol. (Cao, et al., 2017) Links via Pat on my (Telegram channel) thanks! More info is included below about each of these phytonutrients (and ofloxacin is a medication).

Another scientist is also saying VAIDS following CoV jabs leading to shingles or Monkeypox is the real epidemic.

• Top Virologists Blow Whistle: Monkeypox Is Actually COVID Vaccine-Induced ‘Autoimmune Blistering Disease’, (thepeoplesvoice.tv)

Many herbal extracts or foods may be potent against Monkeypox or other infections - including Black Seed oil - which helped me topically and by mouth when I had shingles/sunburn pox in 2022 (following passive exposure CoV jab illness ~ May & Oct/Nov 2021).

Other herbal extracts or foods that might help against Monkeypox include Garlic, which is an Nrf2 promoter and source of cysteine for glutathione production. Pawpaw leaves and mango leaves have shown efficacy and pumpkin leaf and seed and watermelon seeds - likely made into an extract. Olive leaves and stem, Moringa leaf and Nigella sativa, (black seed oil), seeds and leaves. See Table 2 for more plants - 25 are included, and then see Table 3 for the same plant remedies grouped by the symptom cluster that their mechanism of actions may treat. (Sahu, et al., 2023, pdf)

Moringa leaves are used as a food, green veggie, in India, and is available is a powder supplement. It is very rich in vitamin A carotenoids and other phytonutrients. I used about a tablespoon per day of the green powder mixed with cocoa powder in my father’s chocolatey breakfast porridge while caring for him on Hospice - he lasted 16 months on my care after major surgery for CoV injection turbocancer left him partially paralyzed. *Being bedbound is a health risk no matter what else is going on. “Take home question - Is "aggressive care" really helping? as much as pomegranate peel and chocolate porridge might help?” Approximately, 480 days of porridge cooking most mornings - a double boiler is better. **My dad was a quirky guy - my protein rich recipe was based on his own chocolatey routine. (Substack post)

Hospice - dying with dignity, at home. Pros and cons - and chocolate porridge.

Previous posts on this topic have other self-care tips including how I used black seed oil for ‘shingles’ in 2022.

Part 1. "Mpox clade" - our new disease X is still Monkeypox, but elevated IgG4 and VAIDS following CoV jabs may be the real cause of a pox 'epidemic'. "Mpox clade" may have PCR testing & Trojan Horse jabs on the way . . . Do not comply. Clearly, preserving human health is not really the goal. Aug. 17, 2024, (Substack)

Part 2. GoF history on Monkeypox & a better Lysine/arginine ratio reduces risk of shingles or Herpes - and CoV in my own case. Also, food sources of arginine and lysine and spirulina might help herpes too and African geranium. Aug. 19, 2024, (Substack)

Part 3. Mpox jabs are dangerous and may be deadly for people with VAIDS/AIDS or eczema. Passive exposure from jabbed people to those at risk may also be sharing the adverse reaction risk to the unjabbed person. Heart disease and eye conditions are also contraindicated for the Mpox jabs. Aug. 20, 2024, (Substack)

“We have nothing to fear but fear itself.” - Winston Churchill

… and diabolical plots by for-profit depopulators.

Culling for a profit, is killing for a profit, and it often uses tax-payer dollars.

A different Telegram comment shared this comment about the WHO data on Mpox statistics - the main point being - Why are we being frightened with this - the risk is not that great? The risk of Monkeypox is generally not fatal (0.2%) and the group at risk is primarily young adult males, often HIV positive, who are spreading infection through sexual contact. (t.me/deNutrientsChat)

MONKEYPOX UPDATE ATTORNEY

https://t.me/RA_Roehrig/8135 ➡️🐵➡️

🔷 WHO data on Mpox🔷 Hélène Banoun: “And with these data some want to spread fear?" 🔷

After I published many posts about Mpox yesterday and also reported that mainstream reporting is "focused on emergencies," I found some comments by Hélène Banoun that can help calm people down if neighbors or acquaintances become worried. Hélène Banoun is a French pharmacologist and molecular biologist who has studied COVID-19 injections intensively and has worked out their character as "gene therapeutics."

It refers to the WHO publication on data on the spread of Mpox and points out the following:

👉 With the exception of the African continent, according to these data, the occurrence of new "laboratory-confirmed cases" is declining;

👉 The fatality rate is 0.2% between 2022 and 2024;

👉 96.4% of cases affect young men between 29 and 41 years of age and transmission occurs through sexual contact in 83.8% of cases;

👉 Half of those affected are HIV positive.

These data lead Hélène Banoun to ask:

"And with this data some people want to spread fear?"

🔷 Attorney Dr. Brigitte Röhrig via Schultz (t.me/deNutrientsChat)

Taking a risky ‘vaccine’ seems like a greater risk than practicing safe sex or avoiding sexual contact with young adult males, especially those who are HIV positive - it is a known risk factor for Monkeypox. Monkeypox infections cause greater risk during pregnancy for the woman and the fetus. (Sahu, et al., 2023) also via Pat on Telegram.

As I have said in previous posts in this series - “pox” seems to me, to be a symptom of hyperinflammation with poor detoxification and lack of glutathione production.

VAIDS after CoV injections or passive exposure illness is not an imaginary fear - risk is increased and the Mpox injections should be avoided per Geert Vanden Bossche if you got the CoV injections.

. . . and we all have been passively exposed to jabbed people and therefore we all may be at increased risk for adverse reactions to this or other vaccines.

CoV injections and passive exposure to the CoV injected, may have left many of us with VAIDs which would put us in the at risk “HIV+” category regarding Monkeypox risk. That means caution and preventive self-care is likely a sensible idea. Along with avoiding the risky Mpox injections/vaccine. See Geert's video warning on X (or on Rumble directly) - anyone who got CoV injections are being warned against it by Geert Vanden Bossche.

Brenda (X.com/44amazinggrace)

I would add anyone who got passive exposure symptoms too - like me. A lab test for T cell count and type could help show diagnostically if “VAIDS” is a problem - Vaccine Acquired Immune Deficiency Syndrome.

Typical medications used for monkeypox or vaccine injury pox - ‘vaccinia’.

Typical medications used for treating monkeypox include Cidofovir, brincidofovir, and tecovirimat, but no specific medication for monkeypox treatment has been approved by the FDA. Pox “vaccinia” related to vaccine complications can be treated with FDA approved vaccinia immune globulin intravenous (VIGIV), (Sahu, et al., 2023), . . . suggesting that pox like reactions in CoV injected people might respond well to VIGIV. There is a caution against use of VIGIV in people with IgA deficiency or hypersensitivity: “Adverse reactions and side effects: Local injection-site reaction; infusion reaction (not recommended in people with IgA deficiency and possible IgA hypersensitivity)” (Table 1., Sahu, et al., 2023)

“There are no specific monkeypox treatments that have yet received FDA approval. Cidofovir, brincidofovir (a lipidconjugate prodrug of cidofovir), and tecovirimat are just a few of the antiviral medications that have been shown to be effective against MPXV (Table 1). Vaccine-related complications including progressive vaccinia and severe widespread vaccinia can be treated with vaccinia immune globulin intravenous (VIGIV), which has been approved by the Food and Drug Administration (FDA).” […]

“Even though smallpox immunisation is not recommended, those with severe T cell function impairments who have been exposed to the virus may benefit from prophylactic therapy.17” (Sahu, et al., 2023, pdf)

A different comment shared the treatment nitazoxanide:

• Nipping poxviruses in the bud with nitazoxanide, From the Geballe lab, Human Biology Division, July 16, 2018 • By — Y Lim “This study is the first to demonstrate the ability of NTZ to inhibit a poxvirus, and establishes the potential use of NTZ to control poxviral infections.” The mechanism of action is not understood yet. https://www.fredhutch.org/en/news/spotlight/2018/07/hb_hickson_virology.html

Phytonutrient food sources and other benefits

Rosmarinic acid

Rosmarinic acid is available as a supplement and natural food sources include the pine needle like herb called rosemary and the herb known as Lemon Balm (Melissa officinalis). Rosemary is an evergreen, low-to-the-ground shrub which can also be grown as a garden plant.

Quercetrin

Quercitrin is a combination of a quercetin molecule with a rhamnose sugar. “Quercitrin is a glycoside formed from the flavonoid quercetin and the deoxy sugar rhamnose.” (Wikipedia)

St. John’s Wort and Melissa officinalis (Lemon balm) are natural sources of quercitrin and the phytonutrient has been found helpful for colitis/bowel conditions. Melissa officinalis also is a source of rosmarinic acid. Saffron is also on this page but I didn’t see quercitrin in the excerpt. (Quercitrin/ScienceDirect) News for me - quercitrin may also help hair growth. (Kim, et al., 2020) Hair growth and Hashimoto’s thyroiditis aside - a comment from a Substack reader also shared thymus peptides as a potential aid for hair regrowth - improve thyroid function and hair growth may follow.

via Phar Percheron, Iodine for Horses, Aug 20,

Thymus Peptides for Hashimoto's Disease

Myricetin herbal and food sources

1. Ardisia elliptica: A plant extract containing myricitrin, a major phenolic compound, which has been shown to exhibit anticancer effects against liver cancer cells (Hep-G2). *An aquatic plant that has become invasive in Florida. (plants.ifas.ufl.edu/plant-directory/ardisia-elliptica/)

2. Ardisia escallonioides: Another plant extract containing myricitrin, which has been demonstrated to inhibit the growth of liver cancer cells (Hep-G2). *An evergreen shrub native to Florida. Birds like the berries and the flowers attract a type of bee. (fnps.org/plant/ardisia-escallonioides)

3. Chinese bayberry: The fruit, bark, and leaves of Chinese bayberry (Myrica rubra) contain myricitrin, which is used as a flavor modifier in dairy products, snack foods, and beverages. *Highly desired in China for the edible fruit. It is a mountainside tree species that, like Gingko trees, requires a male and a female tree to be near each other in order for pollination of the flowers to occur. (wildlandsplants.com/myrica-rubra)

4. Fruits and vegetables: Myricitrin is also found in various fruits and vegetables, including:

   • Black currant: A significant source of myricitrin, with quantities varying among cultivars.

   • Berries (e.g., goji berry, bog blueberry, cranberry, crowberry, rabbit-eye blueberry): Containing myricitrin in smaller amounts.

   • Other vegetables (e.g., tomatoes, parsley, fennel leaves): With lesser amounts of myricitrin.

Note that myricitrin is a flavonoid extracted from plant sources, and its exact concentration may vary depending on the specific plant material, processing methods, and storage conditions. (Brave AI Summary)

Ofloxacin is available as a medication but has safety cautions

“Ofloxacin is a fluoroquinolone (flor-o-KWIN-o-lone) antibiotic that fights bacteria in the body.

Ofloxacin is used to treat bacterial infections of the skin, lungs, prostate, or urinary tract (bladder and kidneys). Ofloxacin is also used to treat pelvic inflammatory disease and Chlamydia and/or gonorrhea.

Fluoroquinolone antibiotics can cause serious or disabling side effects that may not be reversible. Ofloxacin should be used only for infections that cannot be treated with a safer antibiotic.

Ofloxacin may also be used for purposes not listed in this medication guide.” (drugs.com/mtm/ofloxacin.html)

Adverse risks of fluoroquinolone antibiotics:

“Fluoroquinolones are antibiotics that kill or stop the growth of bacteria¹. However, they carry a risk of serious adverse effects such as Clostridioides difficile infection [*cause of a severe diarrhea condition], tendinopathy, neuropathy, and multiple drug-drug interactions². An FDA safety review found that both oral and injectable fluoroquinolones are associated with disabling side effects involving tendons, muscles, joints, nerves and the central nervous system¹.”

FDA updates warnings for fluoroquinolone antibiotics (1)

Fluoroquinolones (2)

Lemon Balm, St. John’s Wort and rosemary are looking pretty good compared to that. *More on quinolones is included later regarding potential use to treat ALS or other demyelinating conditions. Quinolones also have adverse effect risks, but they have not had fluoride added by humans. (Brave AI Summary) Adding fluoride tends to make a medication seem ‘more effective’ because the body can’t break it down as readily…. leading to toxicity. Quinolones are helpful in ALS because they inhibit COX2, an enzyme that is overactive in ALS - which might be a bad thing for people who have normal activity of the COX2 enzyme. Pomegranate peel would help too then, and that has been part of my own ‘negative side effects’ from use of pomegranate and other herbals - too much COX2 inhibition can act like the salicylate excess.

*Another tip in a comment had been that vagal nerve dysfunction can cause similar symptoms. An older reply gave me a lot of detailed self-care pointers:

The BarefootHealer, Jan 3,

🙏Ta for the mention, unnecessary though😉😊

Based on those results-your cholesterol, Vit D, and calcium levels tell me your body does not seem to be converting to active D (not surprising if there is compromised liver function), I don't know your mitochondrial haplotype but regardless, the serum D is in what I class as the lower range (RDI's are BS unless your in WWII or underdeveloped nation!🤦‍♀️🤦‍♀️). If I were your health professional (which I am not), as part of nutritional and light hygiene, I would be scheduling outside time of 30-40mins, 3x per day. Specifically, before 10am, at midday, and at sunset, for 1 month. Thes will reset circadian clock, improve vagal tone, lower cellular stress, support mitochondrial function. Ultimately aiming for D to be sitting around 60- 80nmls, minimum. And no supps don't cut it in the long run.😉

Breathe work is excellent! Singing, humming all activate vagal tone through the vibratory effect on vocal chords, similar to breathing. And hugging living creatures.🤗

Oh and positive psychology has its merits but there is also a phenomenon of control fatigue and toxic positivity. So don't be too hard on yourself. Your actually dealing with a $#!& load of stuff in your life, and world!

And lower vagal tone means resilience and self control will be lower😉 So dont torture your self esteem unnecessarily.🤗🤗🤗🤷‍♀️BTJMO

Oh and here's the obligatory disclaimer, I am not your doctor, a practicing health professional, yadayda and these are purely my own opinion and in no way are intended as professional medical advice, of course consult your doctor, as your right as a fully functioning adult of sound mind and body!😉🤦‍♀️🤦‍♀️🤦‍♀️

Her reply was to this post:

Overview about the computer modeling study - it focused on compounds known to be potent within Traditional Chinese Medicine:

“Numerous natural products have antiviral properties and have been tested [11]. Traditional Chinese Medicine (TCM) is rich in biological diversity and can be further developed to discover fresh compound candidates. Those therapeutic compounds are primarily prepared from heat-clearing and detoxifying herbs, which are rich in different kinds of alkaloids, terpenes, flavonoids, saponins, and carboxylic acids [11,12]. Computer-aided drug design (CADD) has become more advantageous as compared with the traditional approach of screening in reducing the dissipation of resources in terms of cost, time, and effort through markedly diminishing the quantity of compounds and filtering out hits for further HTS [12].” (Hu, et al., 2023)

Quercitrin for hair follicle support:

“The present study aimed to investigate the molecular mechanism of quercitrin, a major constituent of Hottuynia cordata extract, for its hair growth stimulating activities in cultured human dermal papilla cells (hDPCs). Quercitrin enhanced the cell viability and cellular energy metabolism in cultured hDPCs by stimulating the production of NAD(P)H and mitochondrial membrane potential (ΔΨ). The expression of Bcl2, an essential marker for anagen hair follicle and cell survival, was increased by quercitrin treatment. Quercitrin also increased the cell proliferation marker Ki67. The expression of growth factors—such as bFGF, KGF, PDGF-AA, and VEGF—were increased by quercitrin both in mRNA and protein levels. In addition, quercitrin was found to increase the phosphorylation of Akt, Erk, and CREB in cultured hDPCs, while inhibitors of MAPKs reversed the effects of quercitrin. Finally, quercitrin stimulated hair shaft growth in cultured human hair follicles. Our data obtained from present study are in line with those previously reported and demonstrate that quercitrin is (one of) the active compound(s) of Hottuynia cordata extract which showed hair growth promoting effects. It is strongly suggested that the hair growth stimulating activity of quercitrin was exerted by enhancing the cellular energy metabolism, increasing the production of growth factors via activation of MAPK/CREB signaling pathway.” (Kim, et al., 2020)

Quercitrin is an MAO B inhibitor while quercetin is an MAO A inhibitor. *Study on phytonutrients within tobacco smoke. ???

“It has been reported that five subclasses of natural flavonoids including flavanols, flavanones, flavones, isoflavones, and flavonols show MAO inhibitory activity (Vina et al., 2012). Tobacco flavonoids which inhibit MAO are kaempferol, quercetin, rutin, quercitrin, isoquecitrin, naringenin, naringin, and diosmetin (Table 2 and Fig. 5). In vitro human MAO A and MAO B inhibition, and molecular modeling studies were carried out for kaempferol along with quercetin (Gidaro et al., 2016). Kaempferol and quercetin are both potent, reversible, and selective MAO A inhibitors with IC₅₀ values of 0.525 and 3.98 μM, respectively, but they have no effect on MAO B up to 100 μM. In addition, four natural flavonoids, including quercetin, quercitrin, isoquercitrin, and rutin have been evaluated for MAO B inhibitory and free radical scavenging activities (Lee et al., 2001). […]

Quercetin displayed potent MAO A inhibitory activity, while some MAO B inhibitory activity of lower potency was detected or no MAO B inhibitory activity was observed up to 100 µM (Chimenti et al., 2006; Gidaro et al., 2016; Han et al., 2007) (Table 2). Thus quercetin is a selective MAO A inhibitor based on the combined data from several authors.”

A review of monoamine oxidase (MAO) inhibitors in tobacco or tobacco smoke, Sa Weon Hong, ... Penelope Truman, in NeuroToxicology, 2022; viewable at (Quercitrin/ScienceDirect)

ALS & COX2 inhibition tangent - alkaloids may be protective for ALS, also Alzheimer’s or other neurodegenerative or myelin degrading conditions.

One of the article excerpts is extensive, longer than a typical Science Direct topic page - it is from a 188-page long book chapter called ‘Alkaloids’. Quinoline derivatives may also be protective of the hippocampus by stimulating glutathione production and promoting PPAR gamma. *Quinolines are not quite the same molecularly as flavonoids like quercetin or quercitrin, but they are all considered to be Alkaloids. (Brave AI)

“COX-2 is the target of quinoline derivatives such as compounds 4, 9a, 9b, 9c, 9d, and 9e (CS 1.267–1.272), which inhibited the activity of this enzyme with IC₅₀ values equal to 0.09 μM, 0.08 μM, 0.07 μM, 0.07 μM, 0.05 μM, and 0.04 μM, respectively.⁴⁹⁶ Furthermore, compounds 4a, 5, 6, 7, and 8 (CS 1.273–1.277) inhibited the enzymatic activity of COX-2 with IC₅₀ values equal to 0.1 μM, 0.1 μM, 0.1 μM, 0.08 μM, and 0.07 μM, respectively.⁴⁹⁷ COX-2 catalyzes the conversion of arachidonic acid into prostaglandin G2 (PGG₂), from which PGE2 originates.⁴⁹⁸ It is important here to note that the pathophysiology of MS and ALS, particularly, involves COX-2 and iNOS, which produce superoxide anion and NO, respectively, hence myelin insults.^499–500 In regard to ALS, COX-2 is hyperactive, hence the generation of superoxide anions, neuroinflammation, and production of PGE2, which imposes the release of glutamate⁵⁰¹ and the death of neurons via apoptosis.” […]

“Other quinoline derivatives of neuropharmacological interest are JLK1472, JLK1486, JLK1522, and JLK1535 (CS 1.282–1.285), which protected mouse hippocampal (HT-22) cells from glutamate insults by 90%, 60%, 90%, and 70%, respectively, by increasing glutathione (GSH) and stimulating peroxisome proliferator-activated receptor-γ (PPAR-γ).⁵¹⁰ Additional support to the contention that the quinoline scaffold could be viewed as a beneficial pharmacophore is shown by the fact that the brain itself produces kynurenic acid (CS 1.286) and xanthurenic acid (CS 1.287), which inhibited vesicular glutamate transporter (VGLUT) with Ki values equal to 1.3 μM and 0.1 μM, respectively.^511” (Wiart, 2014; viewable at Quercitrin/ScienceDirect)

Disclaimer: This information is being provided for educational purposes within the guidelines of Fair Use and is not intended to provide individual health guidance.

Reference List

(Cao, et al., 2017) Cao S, Realegeno S, Pant A, Satheshkumar PS, Yang Z. Suppression of Poxvirus Replication by Resveratrol. Front Microbiol. 2017 Nov 17;8:2196. doi: 10.3389/fmicb.2017.02196. PMID: 29204136; PMCID: PMC5698801. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5698801/

(Hu, et al., 2023) Hu X, An S, Chu J, Liang B, Liao Y, Jiang J, Lin Y, Ye L, Liang H. Potential Inhibitors of Monkeypox Virus Revealed by Molecular Modeling Approach to Viral DNA Topoisomerase I. Molecules. 2023 Feb 2;28(3):1444. doi: 10.3390/molecules28031444. PMID: 36771105; PMCID: PMC9919579. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9919579/

(Kim, et al., 2020) Kim J, Kim SR, Choi YH, Shin JY, Kim CD, Kang NG, Park BC, Lee S. Quercitrin Stimulates Hair Growth with Enhanced Expression of Growth Factors via Activation of MAPK/CREB Signaling Pathway. Molecules. 2020 Sep 2;25(17):4004. doi: 10.3390/molecules25174004. PMID: 32887384; PMCID: PMC7504764. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7504764/

(Sahu, et al., 2023, pdf) Sahu, P., Sharma, D., Dash, D., Tripathi, V., Tirkey, R., Tare, H., (2023). Traditional Herbal Medicines as a Complementary Treatment for Monkeypox. Int. J. of Pharmaceutical Quality Assurance. 14. 440-445. 10.25258/ijpqa.14.2.32. https://www.researchgate.net/publication/372425138_Traditional_Herbal_Medicines_As_a_Complementary_Treatment_for_Monkeypox Pdf: https://impactfactor.org/PDF/IJPQA/14/IJPQA,Vol14,Issue2,Article32.pdf

(Quercitrin/ScienceDirect) https://www.sciencedirect.com/topics/neuroscience/quercitrin

(Wiart, 2014) Christophe Wiart, Chapter 1 - Alkaloids, Editor(s): Christophe Wiart, Lead Compounds from Medicinal Plants for the Treatment of Neurodegenerative Diseases, Academic Press, 2014, Pages 1-188, ISBN 9780123983732, https://doi.org/10.1016/B978-0-12-398373-2.00001-7. https://www.sciencedirect.com/science/article/pii/B9780123983732000017 Viewable at (Quercitrin/ScienceDirect)