Returning to your normal programming of nutrients and how great they are.
The brain is made of significant amounts of cholesterol and statins, I seem to recall, are associated with dementia for this reason.
Jennifer, your research and how you described cholesterol as a calcium cap with electrical destructive power coincides with my research of how calcium efflux is increased with emf: https://romanshapoval.substack.com/p/how-flu-is-an-electrical-illness
Don't statins also lower testosterone, and by that factor human growth hormone, which would also not help neurodegeneration?
I lost the statin argument not having any medical standing. I'm helping care for a family member with alzheimers and their neurologist took her off one of the statins and months later we got a message from a lab tech demanding to know why she was taken off the statin.
I brought as much research and studies as the primary care giver/guardian would tolerate in a twenty minute reading session. They seemed unconvinced and ready to be spoonfed whatever pharmacaptured crap a lab tech or bought doc will tell them. It's frustrating but I really do not have any medical standing and can only differ to a system that I no longer trust.
Long story short I stopped my statin a few weeks ago.
Now what should I do? How much magnesium should I take a day? Right now I’m taking 375 mg of mixed OTC magnesium. I have always wondered if I should take more.
Any other recommendations that I could be missing?
Would the use of a water soluble stain instead of a fat soluble statin help reduce the dementia in the brain?
"The prevalence of moderate to severe brain atherosclerosis was 54% among 1905-1914 births, 37% for 1915-1919, 30% for 1920-1924, and 22% for 1925-1930, while moderate/severe arteriolosclerosis ranged from 44% for those born from 1905-1914 to 28% from 1925-1930 (χ2 test: P<0.001 across birth year categories). However, there were no differences in age-standardized prevalence of gross chronic infarcts over time, and microinfarcts were more prevalent with later year of birth."
There are SO many variables here, and in these time frames, that I think it irresponsible journalism to lay it at the feet of one (statins) on the basis of sheer speculation. There is not even a speck of data cited for statin use in these populations, or comparators to non-dementia populations.
Also, why more micro-infarcts? Particularly in an era with MORE aggressive treatment of hypertension?
There is still much to understand about tau protein, amyloid, and dementias.